“Mild” Traumatic Brain Injury (TBI) Doesn’t Exist

brain

Traumatic Brain Injury (TBI) is commonly caused by direct trauma to the skull in which in turn directly interferes with axon connectivity between the brains neural activities through physical motion, change, blunt force, or operational compression. There are various forms of TBI, but most involve the literal shaking and misplacement of the brain and it’s vital parts, leading to hemorrhaging or damage to the brain from such motion—for example, being struck in the head during a boxing match.

Developmental Treatment
There are various forms of physical and psychiatric therapy in efforts to rehabilitate or restore damaged cells, axon behavior and connectivity, and regrowth or repair within the brain. Unfortunately, most of these methods are only in testing phases, and do not always prove successful.
Diffuse Axonal behavior takes place typically after a blow to the head, and in turn disrupts the calcium and iron intake of the brain and it’s axons in efforts to repair itself—a very vital, interrupted process commonly leading to paralysis and even death. Therefore, the most effective therapeutic treatments would, in the future, likely be on a molecular level and involve neurosurgery.

The Science behind the Damage
According to research done by the Perelman School of Medicine at the University of Pennsylvania, as well as a conjointly with a university in the UK, damage to’ the brain and deteriorating or morphing (“retarded”) axon cell behavior can be traced back through blood tests—via SNTF, a protein in the blood which rises after impact or concussion to the brain.
Scientists have recently discovered that another marker for showing signs of deterioration or a likelihood of indefinite TBI damage is a protein called APP also found in the blood stream, but not always.
The dilemma with testing between the two of these through an individual’s blood is that one is weaker and can actually repair itself within the course of several months, while it’s unlike that SNTF levels will improve. This sort of scientific and chemical ‘phenomenon’ makes it all the more difficult to track, make sense of, and try to chemically or biochemically mimic the behavior and enhance or motivate redevelopment of damaged cells or axons due to a concussion—or in this case, Traumatic Brian Injury (TBI).

New Ways of Discovering TBI
Throughout history scientists and doctors alike have relied on CT and MRI scans to reveal concussions, and to what extent or severity and potential of TBI existed. However, this has not always been effective since there are blatant variations of levels of bruising within the brain-predominantly in the instance of a TBI. Since internal bleeding is also rare with concussions, unless under extreme circumstances, it’s arguably difficult to discover said damages and to what velocity via these types of radio imagery scans of the brain and skull.
Microscopic experimentation and intervention in cadavers showed that the axons were in fact disconnected in TBI patients and likely caused the deterioration of not only the brain, but that it was nearly scientifically impossible to stop the degenerative behavior from the source, as the damage and strain had already taken place among neurochemical elements within the brains radio-activities between its axons and neural connectivity.

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